Comman name-SAFED DAAG, PHULERI
PHULERI/ SAFED DAAG/ VlTILIGO is a chronic acquired autoimmune depigmentary skin disorder that cause loss of pigmentation resulting in irregular pale patches in skin. The extent and rate of color loss from vitiligo is unpredictable. It can affect the skin on any part of body. It may also affect hair and the inside of the mouth.
Normally, the color of hair and skin is determined by melanin.
VITILIGO OCCUR WHEN THE MELONOCYTES CELL WHICH PRODUCE MELANIN( RESPONSIBLE FOR SKIN PIGMENTATION) DIE OR UNABLE TO FUNCTION.
Vitiligo is the only skin disease Which does not itch, pain, or give any sort of physical discomfort.
The condition is not life-threatening or contagious. It can be stressful or make feel bad about yourself.
ETIOLOGY
Exact etiology is not known,and May vary from one pattern of diseases to another:
GENETIC:-
· 20-30% of patients give a positive family history.
· Inheritance may be polygenic
AUTOIMMUNE HYPOTHESIS:-
· Vitiligo is frequently associated with autoimmune disorder like alopecia areata and thyroid disorder.
· Antibodies to melanocytes have been also demonstrated.
· Histopathology of early lesion demonstrates presence of lymphocytes in the lesions.
NEUROGENIC HYPOTHESIS:-
· Dermatomal distribution of segmental vitiligo suggest that a toxins, which destroy melanocytes , is released at the nerve endings.
AT THE TIME LESIONS DEVELOP ALONG THE DISTRIBUTION OF PERIPHERAL NERVE CALLED ”ZOSTERIFORM VITILIGO”.
SIGN & SYMPTOMS
The main sign of vitiligo is patchy loss of skin color. Usually, the discoloration first shows on sun-exposed areas, such as the hands, feet, arms, face and lips.
Vitiligo signs include:
· Patchy loss of skin color
· Premature whitening or graying of the hair on your scalp, eyelashes, eyebrows or beard
· Loss of color in the tissues that line the inside of your mouth and nose (mucous membranes)
· Loss of or change in color of the inner layer of the eyeball (retina)
Vitiligo is more noticeable in darker skinned people because of the contrast of white patches Against dark skin.
CAUSE, INCIDENCE, AND RISK FACTOR
Vitiligo appear to occur When immune cells destroy the cell that produce brown pigment, this destruction is thought to be due to autoimmune problem, but the cause is unknown.
AGE FACTOR
Vitiligo may apper any age.
ASSOCIATED WITH OTHER AUTOIMMUNE DISORDER
CUTANEOUS DISORDER
· Alopecia areata
· Halo nevus
· Atopic dermatitis
· Malignant melanoma
· Etc
ENDOCRINE DISORDER
- Diabetes mellitus
- Prenicious anemia
- Adision’s diseases
- Hyperthyroidism
- Hyperparathyroidism
- Etc.
MOST COMMON SITE OF ONSET OF VITILIGO LESION
- LOWER LIMBS (42.5%),
- FOLLOWED BY FACE (27.5%),
- UPPER LIMBS (18.6%),
- FINGER TIPS (7.5%),
- TRUNK (8.6%),
- SCALP (2.5%),
- GENITAL MUCOSA (2.5%) IN THE DESCENDING ORDER OF FREQUENCY
- SITE OF SCRATCHING
TYPES OF VITILIGO (DEPENDING UPON DISCOLOURD PATCHES)
GENERALIZED VITILIGO
With this most common type, called generalized vitiligo, the discolored patches often progress similarly on corresponding body parts (symmetrically).
TYPES -
ACROFACIAL VITILIGO-IN this types lesions are seen periorificially (around eyes and on lips) and acral parts(periungual areas, palm and soles).
LIP TIP VITILIGO- When lips , tips of penis and the vulva and nipples are involved.
SEGMENTAL VITILIGO
Only one side affect ,or( one part of body )This type, called segmental vitiligo, tends to occur at a younger age, progress for a year or two, then stop.
LOCALIZED(FOCAL VITILIGO)
LOCALIZED(FOCAL VITILIGO)
When one or only few areas affected ,This type is called localized (focal) vitiligo.
UNIVERSALIS VITILIGO
UNIVERSALIS VITILIGO
Widespred vitiligo with only few areas of normal pigmentation, it is often associated with multiple endocrinopathies.
VITILIGO VULGARIS
Commonest types of vitiligo,occur after second decade of life . may be slowly or rapidly progressive.
CLINICAL CLASSIFICATION
V1 STAGE-
Active progressive stage
Lesion increases in size
Border increases in size
V2 STAGE-
Quiescent stage
No new lesion developing
Lesions in the stationary in size
Border hyper –pigmented an well defined
V3 STAGE-
Repigmentary stage
Lesions decreases in size
No new lesion developing
Border defined and sign of spontaneous repignentation.
PREVENTION
Lifestyle related –
· Tight fittings, which give marks on the skin, like elastic must be avoided which disturbs the skin blood circulation.
· Scratching, itching must be avoided.
· Any injury will give rise to new patch.
· Plastic and rubber wear should be avoided.
· Plastic Ornaments, Bindi or any Stickers on the skin should be avoided.
Vitiligo diet in order to boost the immune system, as vitiligo is an autoimmune disease and a strong immune system can prevent the spread of the disease.
Diet restriction has much important role in Vitiligo & Leucoderma.
· Curd, Tamarind, Raw Tomato, Raw Onion, Raw garlic, Brinjal, Papaya, Green chili, Pickles, Citrus fruits & Citrus fruit items / juices as oranges, lemon even grapes are strictly restricted.
· Fish and red meat should also be restricted. Oily / spicy food & non vegetarian food intake should be lowered.
· Milk & milk product intake to be lowered (not in case of kids).
· One should not take medicines containing Amla (Gooseberry - a rich source of Vit.C)
· Any eatable made of sodium bi carb, stored sour things, junk food, tinned foods or drinks, chocolates, coffee or cocoa products should not be taken regularly.
HOMOEOPATHIC TREATMENT FOR VITILIGO
The homeopathic remedies given below indicate the therapeutic affinity but this is not a complete and definite guide to the homeopathy treatment of this condition.
The selection of remedy is based upon the theory of individualization and symptoms similarity by using holistic approach.
Several remedies are available to treat that can be selected on the basis of Causes, Sensations, Modalities , & Symptoms of the complaints.
SOME OF THE COMMANLY USED HOMOEOPATHIC MEDICINE FOR VITILIGO
ARSENIC SULPH FLAVUM
SYPHILLINUM
SEPIA
NAT.MUR
ARS ALB
PHOUS
CANTHARIS
HYDROCOTYLE
SULPH
NIT. ACID
CARCINOCIN
MANGI.INDICA
PSORALIA
NUX VOM
BABCHI
IGNATIA
PULS
MEDORRHINUM
LYCO
ARS IOD
BACILLINUM
COBAL. NITRICUM
CUP.AC
CAL.FLOUR
ETC.
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